CRISPR-Cas9 correction of OPA1 c.1334G>A: p.R445H restores
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Functional genomics and the future of iPSCs in disease modeling
CRISPR-AsCas12a Efficiently Corrects a GPR143 Intronic Mutation in
CRISPR/Cas9-mediated A4GALT suppression rescues Fabry disease
IJMS, Free Full-Text
Retinal Ganglion Cells in a Dish: Current Strategies and
Role of Oxidative Stress in Ocular Diseases Associated with
Understanding the molecular basis and pathogenesis of hereditary
CRISPR/Cas9-mediated A4GALT suppression rescues Fabry disease
Optic Atrophy 1 Controls Human Neuronal Development by Preventing
CRISPR-AsCas12a Efficiently Corrects a GPR143 Intronic Mutation in
CRISPR/Cas9-mediated A4GALT suppression rescues Fabry disease
Functional genomics and the future of iPSCs in disease modeling
Characterisation of a novel OPA1 splice variant resulting in
Understanding the molecular basis and pathogenesis of hereditary
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por adulto (o preço varia de acordo com o tamanho do grupo)