CBP-HSF2 structural and functional interplay in Rubinstein-Taybi neurodevelopmental disorder
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Descrição
Schematic illustration of HSF1-HSF2 heterotrimerization as a mechanism
Deletion of Sse1 promotes Cin8-dependent spindle elongation in S phase.
Ultra-Rare Syndromes: The Example of Rubinstein-Taybi Syndrome. - Abstract - Europe PMC
Cdh2 on 293TA cells provides favorable surface for neurite branching
(PDF) CBP-HSF2 structural and functional interplay in Rubinstein-Taybi neurodevelopmental disorder
CBP/EP300-dependent acetylation and stabilization of HSF2 are compromised in Rubinstein-Taybi syndrome
Variations in brain defects result from cellular mosaicism in the activation of heat shock signalling
A conserved PHD finger in the acetyltransferase domain of CBP and p300.
Identification of p35 as a potential target gene for HSF2. (A) Western
CBP-HSF2 structural and functional interplay in Rubinstein-Taybi neurodevelopmental disorder
Stress pathways in neurodevelopmental disorders
Publications
CBP/EP300-dependent acetylation and stabilization of HSF2 are compromised in Rubinstein-Taybi syndrome
CBP/EP300-dependent acetylation and stabilization of HSF2 are compromised in Rubinstein-Taybi syndrome
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