Critical POU domain residues confer Oct4 uniqueness in somatic cell reprogramming
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Descrição
LSD1-mediated demethylation of OCT4 safeguards pluripotent stem cells by maintaining the transcription of PORE-motif-containing genes
Evolutionary Origin of Vertebrate OCT4/POU5 Functions in Supporting Pluripotency
Structural mechanism of LIN28B nucleosome targeting by OCT4 for pluripotency
Critical POU domain residues confer Oct4 uniqueness in somatic cell reprogramming
Evolutionary origin of vertebrate OCT4/POU5 functions in supporting pluripotency
Reprogramming competence of OCT factors is determined by transactivation domains
Reprogramming competence of OCT factors is determined by transactivation domains
Structural mechanism of LIN28B nucleosome targeting by OCT4 for pluripotency
A Late Transition in Somatic Cell Reprogramming Requires Regulators Distinct from the Pluripotency Network - ScienceDirect
The RKRKR motif mutants of Oct4 lose transcriptional activity
Genes, Free Full-Text
A unique Oct4 interface is crucial for reprogramming to pluripotency
Mutation pattern across the POU family and within the OCT4 POU. (A)
Cancers, Free Full-Text
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